The transcription factor Foxg1 regulates telencephalic progenitor proliferation cell autonomously, in part by controlling Pax6 expression levels

Lignende værker

• Controlled overexpression of Pax6 in vivo negatively autoregulates the Pax6 locus, causing cell-autonomous defects of late cortical progenitor proliferation with little effect on cortical arealization
  af: Manuel, Martine, et al.
  Udgivet: (2007)
• The transcription factor Foxg1 regulates the competence of telencephalic cells to adopt subpallial fates in mice
  af: Manuel, Martine, et al.
  Udgivet: (2010)
• Tissue-Specific Actions of Pax6 on Proliferation and Differentiation Balance in Developing Forebrain Are Foxg1 Dependent
  af: Quintana-Urzainqui, Idoia, et al.
  Udgivet: (2018)
• Pax6 controls cerebral cortical cell number by regulating exit from the cell cycle and specifies cortical cell identity by a cell autonomous mechanism
  af: Quinn, Jane C., et al.
  Udgivet: (2007)
• Disruption of Early Events in Thalamocortical Tract Formation in Mice Lacking the Transcription Factors Pax6 or Foxg1
  af: Pratt, Thomas, et al.
  Udgivet: (2002)