Delayed Postnatal Loss of P/Q-Type Calcium Channels Recapitulates the Absence Epilepsy, Dyskinesia, and Ataxia Phenotypes of Genomic Cacna1A Mutations

Lignende værker

• Adult loss of Cacna1a in mice recapitulates childhood absence epilepsy by distinct thalamic bursting mechanisms
  af: Miao, Qing-Long, et al.
  Udgivet: (2020)
• Postnatal Loss of P/Q-Type Channels Confined to Rhombic-Lip-Derived Neurons Alters Synaptic Transmission at the Parallel Fiber to Purkinje Cell Synapse and Replicates Genomic Cacna1a Mutation Phenotype of Ataxia and Seizures in Mice
  af: Maejima, Takashi, et al.
  Udgivet: (2013)
• Absence Epilepsy and Paroxysmal Dyskinesia
  af: J Gordon Millichap
  Udgivet: (2002-12-01)
• Isolated P/Q Calcium Channel Deletion in Layer VI Corticothalamic Neurons Generates Absence Epilepsy
  af: Bomben, Valerie C., et al.
  Udgivet: (2016)
• Hippocampus-related cognitive disorders develop in the absence of epilepsy and ataxia in the heterozygous Cacna1a mutant mice tottering
  af: Akito Nakao, et al.
  Udgivet: (2022-12-01)