Ca(2+)/calmodulin-stimulated PDE1 regulates the beta-catenin/TCF signaling through PP2A B56 gamma subunit in proliferating vascular smooth muscle cells

The phenotypic change of vascular smooth muscle cells (VSMCs), from a “contractile” phenotype to “synthetic” phenotype, is crucial for pathogenic vascular remodeling in vascular diseases such as atherosclerosis and restenosis. Ca(2+)-calmodulin stimulated phosphodiesterase 1 (PDE1) isozymes, includi...

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Detalhes bibliográficos
Main Authors: Jeon, Kye-Im, Jono, Hirofumi, Miller, Clint L., Cai, Yujun, Lim, Soyeon, Liu, Xuan, Gao, Pingjin, Abe, Jun-Ichi, Li, Jian-Dong, Yan, Chen
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3059601/
https://ncbi.nlm.nih.gov/pubmed/21078118
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1742-4658.2010.07908.x
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