Neurofibrillary and neurodegenerative pathology in APP-transgenic mice injected with AAV2-mutant TAU: neuroprotective effects of Cerebrolysin

Alzheimer’s disease (AD) continues to be the most common cause of cognitive and motor alterations in the aging population. Accumulation of amyloid β (Aβ)-protein oligomers and the microtubule associated protein-TAU might be responsible for the neurological damage. We have previously shown that Cereb...

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Bibliographic Details
Main Authors:	Ubhi, Kiren, Rockenstein, Edward, Doppler, Edith, Mante, Michael, Adame, Anthony, Patrick, Christina, Trejo, Margarita, Crews, Leslie, Paulino, Amy, Moessler, Herbert, Masliah, Eliezer
Format:	Artigo
Language:	Inglês
Published:	2009
Subjects:	Article
Online Access:	https://ncbi.nlm.nih.gov/pmc/articles/PMC3049872/ https://ncbi.nlm.nih.gov/pubmed/19252918 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00401-009-0505-4
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Neurofibrillary and neurodegenerative pathology in APP-transgenic mice injected with AAV2-mutant TAU: neuroprotective effects of Cerebrolysin

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