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Oestrogen-mediated protection of experimental autoimmune encephalomyelitis in the absence of Foxp3(+) regulatory T cells implicates compensatory pathways including regulatory B cells
Oestrogen (17β-oestradiol, E(2)) is a highly effective treatment for experimental autoimmune encephalomyelitis (EAE) that may potentiate Foxp3(+) regulatory T (Treg) cells, which in turn limit the expansion of encephalitogenic T-cell specificities. To determine if Treg cells constitute the major non...
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| Main Authors: | , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Blackwell Science Inc
2011
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3044900/ https://ncbi.nlm.nih.gov/pubmed/21091909 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1365-2567.2010.03380.x |
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