Increased fat and skeletal muscle beta-adrenergic receptors but unaltered metabolic and hemodynamic sensitivity to epinephrine in vivo in experimental human thyrotoxicosis.
Based largely on evidence of increased target tissue beta-adrenergic receptor densities and responsiveness in animal and, to a lesser extent, human tissues, it is often assumed that thyroid hormone excess results in increased sensitivity to catecholamines in vivo, thus explaining several clinical ma...
Guardado en:
| Publicado en: | J Clin Invest |
|---|---|
| Autores principales: | , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Society for Clinical Investigation
1989
|
| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC303751/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2537849/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI113961 |
| Etiquetas: |
Sin Etiquetas, Sea el primero en etiquetar este registro!
|