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Evasion of Cellular Antiviral Responses by Human Cytomegalovirus TRS1 and IRS1
During infection with human cytomegalovirus (HCMV), cellular protein synthesis continues even as viral proteins are being synthesized in abundance. Thus, HCMV may have a mechanism for counteracting host cell antiviral pathways that act by shutting off translation. Consistent with this view, HCMV inf...
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| Hoofdauteurs: | , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Society for Microbiology
2004
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC303427/ https://ncbi.nlm.nih.gov/pubmed/14671101 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.78.1.197-205.2004 |
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