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Evasion of Cellular Antiviral Responses by Human Cytomegalovirus TRS1 and IRS1

During infection with human cytomegalovirus (HCMV), cellular protein synthesis continues even as viral proteins are being synthesized in abundance. Thus, HCMV may have a mechanism for counteracting host cell antiviral pathways that act by shutting off translation. Consistent with this view, HCMV inf...

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書目詳細資料
Main Authors: Child, Stephanie J., Hakki, Morgan, De Niro, Katherine L., Geballe, Adam P.
格式: Artigo
語言:Inglês
出版: American Society for Microbiology 2004
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC303427/
https://ncbi.nlm.nih.gov/pubmed/14671101
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.78.1.197-205.2004
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