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Defective trafficking and function of K(ATP) channels caused by a sulfonylurea receptor 1 mutation associated with persistent hyperinsulinemic hypoglycemia of infancy
The ATP-sensitive potassium channel (K(ATP)) regulates insulin secretion in pancreatic β cells. Loss of functional K(ATP) channels because of mutations in either the SUR1 or Kir6.2 channel subunit causes persistent hyperinsulinemic hypoglycemia of infancy (PHHI). We investigated the molecular mechan...
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| Главные авторы: | , , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
The National Academy of Sciences
2001
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC30234/ https://ncbi.nlm.nih.gov/pubmed/11226335 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.051499698 |
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