MyD88 Deficiency Leads to Decreased NK Cell Gamma Interferon Production and T Cell Recruitment during Chlamydia muridarum Genital Tract Infection, but a Predominant Th1 Response and Enhanced Monocytic Inflammation Are Associated with Infection Resolution

Lignende værker

• CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection()
  af: Frazer, Lauren C., et al.
  Udgivet: (2013)
• Type I Interferon Signaling Exacerbates Chlamydia muridarum Genital Infection in a Murine Model
  af: Nagarajan, Uma M., et al.
  Udgivet: (2008)
• Stimulator of IFN Gene Is Critical for Induction of IFN-β during Chlamydia muridarum Infection
  af: Prantner, Daniel, et al.
  Udgivet: (2010)
• Significant role of IL-1 signaling, but limited role of inflammasome activation in oviduct pathology during Chlamydia muridarum genital infection()
  af: Nagarajan, Uma M., et al.
  Udgivet: (2012)
• Critical Role for Interleukin-1β (IL-1β) during Chlamydia muridarum Genital Infection and Bacterial Replication-Independent Secretion of IL-1β in Mouse Macrophages
  af: Prantner, Daniel, et al.
  Udgivet: (2009)