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Ca(2+) paradox injury mediated through TRPC channels in mouse ventricular myocytes

BACKGROUND AND PURPOSE: The Ca(2+) paradox is an important phenomenon associated with Ca(2+) overload-mediated cellular injury in myocardium. The present study was undertaken to elucidate molecular and cellular mechanisms for the development of the Ca(2+) paradox. EXPERIMENTAL APPROACH: Fluorescence...

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Detalhes bibliográficos
Main Authors: Kojima, Akiko, Kitagawa, Hirotoshi, Omatsu-Kanbe, Mariko, Matsuura, Hiroshi, Nosaka, Shuichi
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2010
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3010579/
https://ncbi.nlm.nih.gov/pubmed/20718730
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2010.00986.x
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