Lack of commensal flora in H. pylori-infected INS-GAS mice reduces gastritis and delays intraepithelial neoplasia

BACKGROUND & AIMS: Transgenic, insulin–gastrin (INS–GAS) mice have high circulating levels of gastrin. On a FVB/N background, these mice develop spontaneous atrophic gastritis and gastrointestinal intraepithelial neoplasia (GIN) with 80% prevalence 6 months after Helicobacter pylori infection. G...

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Huvudupphovsmän: Lofgren, Jennifer L., Whary, Mark T., Ge, Zhongming, Muthupalani, Sureshkumar, Taylor, Nancy S., Mobley, Melissa, Potter, Amanda, Varro, Andrea, Eibach, Daniel, Suerbaum, Sebastian, Wang, Timothy C., Fox, James G.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2010
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3006487/
https://ncbi.nlm.nih.gov/pubmed/20950613
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1053/j.gastro.2010.09.048
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