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The timing of phasic transmitter release is Ca(2+)-dependent and lacks a direct influence of presynaptic membrane potential
Ca(2+) influx through voltage-gated Ca(2+) channels and the resulting elevation of intracellular Ca(2+) concentration, [Ca(2+)](i), triggers transmitter release in nerve terminals. However, it is controversial whether in addition to the opening of Ca(2+) channels, membrane potential directly affects...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2003
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC299956/ https://ncbi.nlm.nih.gov/pubmed/14630950 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2433276100 |
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