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Translocation of synapsin I in response to depolarization of isolated nerve terminals.

Depolarization of isolated nerve terminals (synaptosomes) has been shown to stimulate neurotransmitter release and to increase the phosphorylation state of a number of proteins, including synapsin I, in a Ca2+-dependent manner. Synapsin I, a prominent nerve terminal phosphoprotein, interacts with th...

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Detalhes bibliográficos
Main Authors: Sihra, T S, Wang, J K, Gorelick, F S, Greengard, P
Formato: Artigo
Idioma:Inglês
Publicado em: 1989
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC298224/
https://ncbi.nlm.nih.gov/pubmed/2510160
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