Reduced miR-146a Increases Prostaglandin E(2) in Chronic Obstructive Pulmonary Disease Fibroblasts

Rationale: Persistent inflammation plays a major role in chronic obstructive pulmonary disease (COPD) pathogenesis, but its mechanisms are incompletely defined. Overproduction of the inflammatory mediator prostaglandin (PG) E(2) by COPD fibroblasts contributes to reduced repair function. Objectives:...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Sato, Tadashi, Liu, Xiangde, Nelson, Amy, Nakanishi, Masanori, Kanaji, Nobuhiro, Wang, Xingqi, Kim, Miok, Li, Yingji, Sun, Jianhong, Michalski, Joel, Patil, Amol, Basma, Hesham, Holz, Olaf, Magnussen, Helgo, Rennard, Stephen I.
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Thoracic Society 2010
Θέματα:
A. Chronic Obstructive Pulmonary Disease
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2970844/
https://ncbi.nlm.nih.gov/pubmed/20522791
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1164/rccm.201001-0055OC
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