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Neuropsychological Deficits in Huntington’s Disease Gene Carriers and Correlates of Early “Conversion”

The authors examined whether the baseline cognitive functioning of 21 clinically normal huntingtin mutation carriers who developed manifest Huntington’s disease on follow-up differed from that of 49 mutation carriers who remain asymptomatic over the same period in a longitudinal study. One hundred t...

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Autori principali: Brandt, Jason, Inscore, Anjeli B., Ward, Julianna, Shpritz, Barnett, Rosenblatt, Adam, Margolis, Russell L., Ross, Christopher A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2008
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2966303/
https://ncbi.nlm.nih.gov/pubmed/19196932
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1176/appi.neuropsych.20.4.466
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