Glycolytic inhibition and calcium overload as consequences of exogenously generated free radicals in rabbit hearts.

Free radicals have been implicated in the pathogenesis of reperfusion injury, but it is unclear how they exert their deleterious effects on cellular metabolism. Several lines of indirect evidence suggest that free radicals elevate intracellular Ca2+ concentration ([Ca2+]i) and inhibit glycolysis as...

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Publié dans:	J Clin Invest
Auteurs principaux:	Corretti, M C, Koretsune, Y, Kusuoka, H, Chacko, V P, Zweier, J L, Marban, E
Format:	Artigo
Langue:	Inglês
Publié:	American Society for Clinical Investigation 1991
Sujets:	Research Article
Accès en ligne:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC295509/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1653271/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI115361
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