Glycolytic inhibition and calcium overload as consequences of exogenously generated free radicals in rabbit hearts.
Free radicals have been implicated in the pathogenesis of reperfusion injury, but it is unclear how they exert their deleterious effects on cellular metabolism. Several lines of indirect evidence suggest that free radicals elevate intracellular Ca2+ concentration ([Ca2+]i) and inhibit glycolysis as...
Uloženo v:
| Vydáno v: | J Clin Invest |
|---|---|
| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
1991
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC295509/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1653271/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI115361 |
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