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Intra-islet insulin permits glucose to directly suppress pancreatic A cell function.

Inhibition of pancreatic glucagon secretion during hyperglycemia could be mediated by (a) glucose, (b) insulin, (c) somatostatin, or (d) glucose in conjunction with insulin. To determine the role of these factors in the mediation of glucagon suppression, we injected alloxan while clamping the arteri...

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Bibliografski detalji
Glavni autori: Greenbaum, C J, Havel, P J, Taborsky, G J, Klaff, L J
Format: Artigo
Jezik:Inglês
Izdano: 1991
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC295460/
https://ncbi.nlm.nih.gov/pubmed/1679440
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