Heat acclimation provides sustained improvement in functional recovery and attenuates apoptosis after traumatic brain injury

Heat acclimation (HA) offers functional neuroprotection in mice after traumatic brain injury (TBI). This study further characterizes endogenous neuroprotection acquired by HA (34±1°C, 30 d) after TBI. We establish here the ability of HA to induce sustained functional benefits and to reduce activatio...

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Bibliographic Details
Main Authors: Umschwief, Gali, Shein, Na'ama A, Alexandrovich, Alexander G, Trembovler, Victoria, Horowitz, Michal, Shohami, Esther
Format: Artigo
Language:Inglês
Published: Nature Publishing Group 2010
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Original Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC2949134/
https://ncbi.nlm.nih.gov/pubmed/19904288
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jcbfm.2009.234
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