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H(2)O(2) alters rat cardiac sarcomere function and protein phosphorylation through redox signaling
ROS, such as H(2)O(2), are a component of pathological conditions in many organ systems and have been reported to be elevated in cardiac pathophysiology. The experiments presented here test the hypothesis that H(2)O(2) induces alterations in cardiac myofilament function by the posttranslational modi...
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| Huvudupphovsmän: | , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
American Physiological Society
2010
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2944474/ https://ncbi.nlm.nih.gov/pubmed/20562337 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00050.2010 |
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