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Endogenous nitric oxide enhances prostaglandin production in a model of renal inflammation.
The interaction between nitric oxide (NO) and cyclooxygenase (COX) was studied in a rabbit model of renal inflammation, the ureteral obstructed hydronephrotic kidney (HNK). Ex vivo perfusion of the HNK but not the control kidney (e.g., unobstructed contralateral kidney, CLK), led to a time-dependent...
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| Vydáno v: | J Clin Invest |
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| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
1994
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC294301/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7514189/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI117185 |
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