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Endogenous nitric oxide enhances prostaglandin production in a model of renal inflammation.
The interaction between nitric oxide (NO) and cyclooxygenase (COX) was studied in a rabbit model of renal inflammation, the ureteral obstructed hydronephrotic kidney (HNK). Ex vivo perfusion of the HNK but not the control kidney (e.g., unobstructed contralateral kidney, CLK), led to a time-dependent...
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| 發表在: | J Clin Invest |
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| Main Authors: | , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
American Society for Clinical Investigation
1994
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC294301/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7514189/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI117185 |
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