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17β-Estradiol Rapidly Increases K(ATP) Activity in GnRH via a Protein Kinase Signaling Pathway
17β-Estradiol (E2) both inhibits and excites GnRH neurons via presynaptic as well as postsynaptic mechanisms. Although it has been demonstrated that E2 can alter the excitability of GnRH neurons via direct actions, the intracellular signaling cascades mediating these actions are not well understood....
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| Hauptverfasser: | , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
The Endocrine Society
2010
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2940490/ https://ncbi.nlm.nih.gov/pubmed/20660067 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/en.2010-0177 |
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