Insulin-like Growth Factor–1 Levels Contribute to the Development of Bacterial Translocation in Sepsis

Rationale: Many lines of evidence point toward the gastrointestinal (GI) tract in the pathophysiology of organ dysfunction in sepsis. Splanchnic hypoperfusion during sepsis leads to enterocyte apoptosis, diminished barrier function, and release of bacterial products. Sepsis lowers levels of insulin-...

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Detalhes bibliográficos
Principais autores: Hunninghake, Gary W., Doerschug, Kevin C., Nymon, Amanda B., Schmidt, Gregory A., Meyerholz, David K., Ashare, Alix
Formato: Artigo
Idioma:Inglês
Publicado em: American Thoracic Society 2010
Assuntos:
D. Critical Care
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2937242/
https://ncbi.nlm.nih.gov/pubmed/20413631
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1164/rccm.200911-1757OC
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