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Insulin-like Growth Factor–1 Levels Contribute to the Development of Bacterial Translocation in Sepsis
Rationale: Many lines of evidence point toward the gastrointestinal (GI) tract in the pathophysiology of organ dysfunction in sepsis. Splanchnic hypoperfusion during sepsis leads to enterocyte apoptosis, diminished barrier function, and release of bacterial products. Sepsis lowers levels of insulin-...
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| Principais autores: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Thoracic Society
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2937242/ https://ncbi.nlm.nih.gov/pubmed/20413631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1164/rccm.200911-1757OC |
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