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Bidirectional Neuron–Glia Interactions Triggered by Deficiency of Glutamate Uptake at Spinal Sensory Synapses

Bidirectional interactions between neurons and glial cells are crucial to the genesis of pathological pain. The mechanisms regulating these interactions and the role of this process in relaying synaptic input in the spinal dorsal horn remain to be established. We studied the role of glutamate transp...

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Autors principals: Nie, Hui, Zhang, Haijun, Weng, Han-Rong
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2010
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2934930/
https://ncbi.nlm.nih.gov/pubmed/20554846
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00282.2010
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