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Impaired Glial Glutamate Uptake Induces Extrasynaptic Glutamate Spillover in the Spinal Sensory Synapses of Neuropathic Rats

Glial cell dysfunction and excessive glutamate receptor activation in spinal dorsal horn neurons are hallmark mechanisms of pathological pain. The way in which glial cell dysfunction leads to excessive glutamate receptor activation in the spinal sensory synapses remains unknown. We and others recent...

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Detalhes bibliográficos
Main Authors: Nie, Hui, Weng, Han-Rong
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2010
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2867569/
https://ncbi.nlm.nih.gov/pubmed/20220084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00013.2010
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