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Impaired Glial Glutamate Uptake Induces Extrasynaptic Glutamate Spillover in the Spinal Sensory Synapses of Neuropathic Rats
Glial cell dysfunction and excessive glutamate receptor activation in spinal dorsal horn neurons are hallmark mechanisms of pathological pain. The way in which glial cell dysfunction leads to excessive glutamate receptor activation in the spinal sensory synapses remains unknown. We and others recent...
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| Autores principales: | , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Physiological Society
2010
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| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2867569/ https://ncbi.nlm.nih.gov/pubmed/20220084 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00013.2010 |
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