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Increased Store-Operated Ca(2+) Entry in Skeletal Muscle with Reduced Calsequestrin-1 Expression

Store-operated Ca(2+) entry (SOCE) contributes to Ca(2+) handling in normal skeletal muscle function, as well as the progression of muscular dystrophy and sarcopenia, yet the mechanisms underlying the change in SOCE in these states remain unclear. Previously we showed that calsequestrin-1 (CSQ1) par...

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מידע ביבליוגרפי
Main Authors: Zhao, Xiaoli, Min, Choon Kee, Ko, Jae-Kyun, Parness, Jerome, Kim, Do Han, Weisleder, Noah, Ma, Jianjie
פורמט: Artigo
שפה:Inglês
יצא לאור: The Biophysical Society 2010
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2931717/
https://ncbi.nlm.nih.gov/pubmed/20816068
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bpj.2010.06.050
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