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Overexpression of SK3 Channels Dampens Uterine Contractility to Prevent Preterm Labor in Mice
The mechanisms that control the timing of labor have yet to be fully characterized. In a previous study, the overexpression of small conductance calcium-activated K(+) channel isoform 3 in transgenic mice, Kcnn3(tm1Jpad)/Kcnn3(tm1Jpad) (also known as SK3(T/T)), led to compromised parturition, which...
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| 主要な著者: | , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2008
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2930016/ https://ncbi.nlm.nih.gov/pubmed/18305226 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1095/biolreprod.107.066423 |
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