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The Matricellular Protein CCN1/CYR61 Induces Fibroblast Senescence and Restricts Fibrosis in Cutaneous Wound Healing

Cellular senescence is a recognised mechanism of tumor suppression; however, its contribution to other pathologies is not well understood. We show that the matricellular protein CCN1/CYR61, which is dynamically expressed at sites of wound repair, can induce fibroblast senescence through its cell adh...

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Autori principali: Jun, Joon-Il, Lau, Lester F.
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2010
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2919364/
https://ncbi.nlm.nih.gov/pubmed/20526329
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncb2070
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