Cardiomyocytes with disrupted CFTR function require CaMKII and Ca(2+)-activated Cl(−) channel activity to maintain contraction rate

The physiological role of the cystic fibrosis transmembrane conductance regulator (CFTR) in cardiomyocytes remains unclear. Using spontaneously beating neonatal ventricular cardiomyocytes from wild-type (WT) or CFTR knockout (KO) mice, we examined the role of CFTR in the modulation of cardiomyocyte...

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Autors principals: Sellers, Zachary M, De Arcangelis, Vania, Xiang, Yang, Best, Philip M
Format: Artigo
Idioma:Inglês
Publicat: Blackwell Science Inc 2010
Matèries:
Cardiovascular
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2915517/
https://ncbi.nlm.nih.gov/pubmed/20442264
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2010.188334
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