A20 inhibits post-angioplasty restenosis by blocking macrophage trafficking and decreasing adventitial neovascularization

OBJECTIVE: Neointimal hyperplasia is an inflammatory and proliferative process that occurs as a result of injury to the vessel wall. We have shown that the homeostatic protein A20 prevents neointimal hyperplasia by affecting endothelial cell (EC) and smooth muscle cell (SMC) responses to injury. In...

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Detalhes bibliográficos
Main Authors: Damrauer, Scott M., Fisher, Mark D., Wada, Hiromi, Siracuse, Jeffrey J., da Silva, Cleide G., Moon, Karam, Csizmadia, Eva, Maccariello, Elizabeth R., Patel, Virendra I., Studer, Peter, Essayagh, Sanah, Aird, William C., Daniel, Soizic, Ferran, Christiane
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2914135/
https://ncbi.nlm.nih.gov/pubmed/20430393
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.atherosclerosis.2010.03.029
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