Comparative Functional Genomics Analysis of NNK Tobacco-Carcinogen Induced Lung Adenocarcinoma Development in Gprc5a-Knockout Mice

Lignende værker

• A Gprc5a Tumor Suppressor Loss of Expression Signature Is Conserved, Prevalent, and Associated with Survival in Human Lung Adenocarcinomas
  af: Humam Kadara, et al.
  Udgivet: (2010-06-01)
• A Gprc5a Tumor Suppressor Loss of Expression Signature Is Conserved, Prevalent, and Associated with Survival in Human Lung Adenocarcinomas
  af: Kadara, Humam, et al.
  Udgivet: (2010)
• Gprc5a deletion enhances the transformed phenotype in normal and malignant lung epithelial cells by eliciting persistent Stat3 signaling induced by autocrine Lif
  af: Chen, Yulong, et al.
  Udgivet: (2010)
• Knockout of the Tumor suppressor gene Gprc5a in mice leads to NF-κB activation in airway epithelium and promotes lung inflammation and tumorigenesis
  af: Deng, Jiong, et al.
  Udgivet: (2010)
• Enhancement of lung tumorigenesis in a Gprc5a Knockout mouse by chronic extrinsic airway inflammation
  af: Barta, Peter, et al.
  Udgivet: (2012)