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Synaptotoxicity of Alzheimer Beta Amyloid Can Be Explained by Its Membrane Perforating Property

The mechanisms that induce Alzheimer's disease (AD) are largely unknown thereby deterring the development of disease-modifying therapies. One working hypothesis of AD is that Aβ excess disrupts membranes causing pore formation leading to alterations in ionic homeostasis. However, it is largely...

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Detalhes bibliográficos
Main Authors: Sepulveda, Fernando J., Parodi, Jorge, Peoples, Robert W., Opazo, Carlos, Aguayo, Luis G.
Formato: Artigo
Idioma:Inglês
Publicado em: Public Library of Science 2010
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2910737/
https://ncbi.nlm.nih.gov/pubmed/20676404
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0011820
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