Low doses of LPS and minimally oxidized LDL cooperatively activate macrophages via NF-kappaB and AP-1: Possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia

RATIONALE: Oxidized low-density lipoprotein (LDL) is an important determinant of inflammation in atherosclerotic lesions. It has also been documented that certain chronic infectious diseases, such as periodontitis and chlamydial infection, exacerbate clinical manifestations of atherosclerosis. In ad...

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Detalhes bibliográficos
Main Authors: Wiesner, Philipp, Choi, Soo-Ho, Almazan, Felicidad, Benner, Christopher, Huang, Wendy, Diehl, Cody J., Gonen, Ayelet, Butler, Susan, Witztum, Joseph L., Glass, Christopher K., Miller, Yury I.
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2904601/
https://ncbi.nlm.nih.gov/pubmed/20489162
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.110.218420
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