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Damage to the gastric epithelium activates cellular bicarbonate secretion via SLC26A9 Cl(−)/HCO(3)(−) exchange

Gastric surface pH (pH(o)) transiently increases in response to focal epithelial damage. The sources of that increase, either from paracellular leakage of interstitial fluid or transcellular acid/base fluxes, have not been determined. Using in vivo microscopy approaches we measured pH(o) with Cl-NER...

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Autores principales: Demitrack, Elise S., Soleimani, Manoocher, Montrose, Marshall H.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Physiological Society 2010
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2904109/
https://ncbi.nlm.nih.gov/pubmed/20413716
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00037.2010
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