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Hypercapnia causes cellular oxidation and nitrosation in addition to acidosis: implications for CO(2) chemoreceptor function and dysfunction

Cellular mechanisms of CO(2) chemoreception are discussed and debated in terms of the stimuli produced during hypercapnic acidosis and their molecular targets: protons generated by the hydration of CO(2) and dissociation of carbonic acid, which target membrane-bound proteins and lipids in brain stem...

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Bibliografske podrobnosti
Glavni avtor: Dean, Jay B.
Format: Artigo
Jezik:Inglês
Izdano: American Physiological Society 2010
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC2886689/
https://ncbi.nlm.nih.gov/pubmed/20150563
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.01337.2009
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