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Hypercapnia causes cellular oxidation and nitrosation in addition to acidosis: implications for CO(2) chemoreceptor function and dysfunction
Cellular mechanisms of CO(2) chemoreception are discussed and debated in terms of the stimuli produced during hypercapnic acidosis and their molecular targets: protons generated by the hydration of CO(2) and dissociation of carbonic acid, which target membrane-bound proteins and lipids in brain stem...
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| Autor principal: | |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2010
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2886689/ https://ncbi.nlm.nih.gov/pubmed/20150563 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.01337.2009 |
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