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Disruption of Sarcolemmal ATP sensitive potassium Channel Activity Impairs the Cardiac Response to Systolic Overload

Sarcolemmal ATP sensitive potassium Channels (K(ATP)) act as metabolic sensors that facilitate adaptation of the left ventricle (LV) to changes in energy requirements. This study examined the mechanism by which K(ATP) dysfunction impairs the LV response to stress using transgenic mouse strains with...

詳細記述

保存先:
書誌詳細
主要な著者: Hu, Xinli, Xu, Xin, Huang, Yimin, Fassett, John, Flagg, Thomas P., Zhang, Ying, Nichols, Colin G., Bache, Robert J., Chen, Yingjie
フォーマット: Artigo
言語:Inglês
出版事項: 2008
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2877276/
https://ncbi.nlm.nih.gov/pubmed/18802029
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.107.170795
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