Effects of neuronal nitric oxide synthase inhibition on resting and exercising hindlimb muscle blood flow in the rat

Nitric oxide (NO) derived from endothelial NO synthase (eNOS) is an integral mediator of vascular control during muscle contractions. However, it is not known whether neuronal NOS (nNOS)-derived NO regulates tissue hyperaemia in healthy subjects, particularly during exercise. We tested the hypothesi...

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Bibliografiska uppgifter
Huvudupphovsmän: Copp, Steven W, Hirai, Daniel M, Schwagerl, Peter J, Musch, Timothy I, Poole, David C
Materialtyp: Artigo
Språk:Inglês
Publicerad: Blackwell Science Inc 2010
Ämnen:
Skeletal Muscle and Exercise
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2872736/
https://ncbi.nlm.nih.gov/pubmed/20176629
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2009.183723
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