Translocation of Amyloid Precursor Protein C-terminal fragment(s) to the nucleus precedes neuronal death due to thiamine deficiency induced mild impairment of oxidative metabolism

Ähnliche Einträge

• The γ-secretase-cleaved C-terminal fragment of amyloid precursor protein mediates signaling to the nucleus
  von: Gao, Yuehua, et al.
  Veröffentlicht: (2001)
• Novel neuritic clusters with accumulations of amyloid precursor protein and amyloid precursor-like protein 2 immunoreactivity in brain regions damaged by thiamine deficiency.
  von: Calingasan, N. Y., et al.
  Veröffentlicht: (1996)
• Thiamine deficiency induces oxidative stress and exacerbates the plaque pathology in Alzheimer’s mouse model
  von: Karuppagounder, Saravanan S., et al.
  Veröffentlicht: (2008)
• Presenilin complexes with the C-terminal fragments of amyloid precursor protein at the sites of amyloid β-protein generation
  von: Xia, Weiming, et al.
  Veröffentlicht: (2000)
• Extracellular vesicles: where the amyloid precursor protein carboxyl‐terminal fragments accumulate and amyloid‐β oligomerizes
  von: Pérez‐González, Rocío, et al.
  Veröffentlicht: (2020)