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Genetic Ablation of Luteinizing Hormone Receptor Improves the Amyloid Pathology in a Mouse Model of Alzheimer Disease
Amyloid-β peptide (Aβ) plays an essential pathophysiological role in Alzheimer disease (AD) and elevation of luteinizing hormone (LH) levels during aging has been implicated in its pathogenesis. To assess the effect of LH receptor deficiency on Aβ accumulation, we generated a bigenic mouse model APP...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2869088/ https://ncbi.nlm.nih.gov/pubmed/20142765 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/NEN.0b013e3181d072cf |
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