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Neuroprotection with Delayed Calpain Inhibition after Transient Forebrain Ischemia
OBJECTIVE: Delayed neurodegeneration after transient global brain ischemia offers a therapeutic window for inhibiting molecular injury mechanisms. One such mechanism is calpain-mediated proteolysis, which peaks 24 to 48 hours after transient forebrain ischemia in rats. This study tests the hypothesi...
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| 主要な著者: | , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2008
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2867247/ https://ncbi.nlm.nih.gov/pubmed/20449914 |
| タグ: |
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