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Hexosamine Flux, the O-GlcNAc Modification, and the Development of Insulin Resistance in Adipocytes
Excess flux through the hexosamine biosynthesis pathway in adipocytes is a fundamental cause of “glucose toxicity” and the development of insulin resistance that leads to type II diabetes. Adipose tissue-specific elevation in hexosamine flux in animal models recapitulates whole-body insulin-resistan...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2855202/ https://ncbi.nlm.nih.gov/pubmed/19799964 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mce.2009.09.022 |
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