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CHRONIC INTERMITTENT HYPOXIA AUGMENTS CHEMOREFLEX CONTROL OF SYMPATHETIC ACTIVITY: ROLE OF THE ANGIOTENSIN II TYPE 1 RECEPTOR
Chronic exposure to intermittent hypoxia (CIH) increases carotid sinus nerve activity in normoxia and in response to acute hypoxia. We hypothesized that CIH augments basal and chemoreflex-stimulated sympathetic outflow through an angiotensin receptor-dependent mechanism. Rats were exposed to CIH for...
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| Main Authors: | , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
2010
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2846996/ https://ncbi.nlm.nih.gov/pubmed/20153844 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.resp.2010.02.003 |
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