A deficit of functional GABAA receptors in neurons of β3 subunit knockout mice

Mice whose γ-aminobutyric acid type A (GABA(A)) β(3) subunit gene is inactivated (‘β(3) knockout mice’) have been previously shown to have epilepsy, hypersensitive behavior, cleft palate, and a high incidence of neonatal mortality. In this study, we analyze whole-cell responses to GABA in neurons fr...

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Huvudupphovsmän: Krasowski, Matthew D., Rick, Caroline E., Harrison, Neil L., Firestone, Leonard L., Homanics, Gregg E.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 1998
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2846962/
https://ncbi.nlm.nih.gov/pubmed/9486477
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