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The Transgenic Overexpression of α-Synuclein and Not Its Related Pathology Associates with Complex I Inhibition
α-Synuclein (αS) is a protein involved in the cytopathology and genetics of Parkinson disease and is thought to affect mitochondrial complex I activity. Previous studies have shown that mitochondrial toxins and specifically inhibitors of complex I activity enhance αS pathogenesis. Here we show that...
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| Autori principali: | , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Biochemistry and Molecular Biology
2010
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2844181/ https://ncbi.nlm.nih.gov/pubmed/20053987 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.061051 |
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