Tumor necrosis factor-α and apoptosis signal-regulating kinase 1 control reactive oxygen species release, mitochondrial autophagy and c-Jun N-terminal kinase/p38 phosphorylation during necrotizing enterocolitis

BACKGROUND: Oxidative stress and inflammation may contribute to the disruption of the protective gut barrier through various mechanisms; mitochondrial dysfunction resulting from inflammatory and oxidative injury may potentially be a significant source of apoptosis during necrotizing enterocolitis (N...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Baregamian, Naira, song, Jun, Bailey, C. Eric, Papaconstantinou, John, Evers, B. Mark, Chung, Dai H
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Landes Bioscience 2009
Aiheet:
Research Paper
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2835918/
https://ncbi.nlm.nih.gov/pubmed/20716917
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