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SIX1 acts synergistically with TBX18 in mediating ureteral smooth muscle formation
Dysfunction of the ureter often leads to urine flow impairment from the kidney to the bladder, causing dilation of the ureter and/or renal pelvis. Six1 is a crucial regulator of renal development: mutations in human SIX1 cause branchio-oto-renal (BOR) syndrome and Six1(–/–) mice exhibit renal agenes...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Company of Biologists
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2827686/ https://ncbi.nlm.nih.gov/pubmed/20110314 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/dev.045757 |
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