Enhanced thrombin generation and platelet binding on herpes simplex virus-infected endothelium.

Atherosclerotic lesions have been reported to contain herpes simplex virus 1 (HSV-1) genomic material. This, and other previous evidence, suggests that latent viral infection may be an atherogenic trigger. Moreover, active HSV-1 lesions manifest marked fibrin deposition in microvessels. In this repo...

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Pubblicato in:	Proc Natl Acad Sci U S A
Autori principali:	Visser, M R, Tracy, P B, Vercellotti, G M, Goodman, J L, White, J G, Jacob, H S
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	National Academy of Sciences 1988
Soggetti:	Research Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC282402/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2847155/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.85.21.8227
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