Enhanced thrombin generation and platelet binding on herpes simplex virus-infected endothelium.
Atherosclerotic lesions have been reported to contain herpes simplex virus 1 (HSV-1) genomic material. This, and other previous evidence, suggests that latent viral infection may be an atherogenic trigger. Moreover, active HSV-1 lesions manifest marked fibrin deposition in microvessels. In this repo...
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| Veröffentlicht in: | Proc Natl Acad Sci U S A |
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| Hauptverfasser: | , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
National Academy of Sciences
1988
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| Schlagworte: | |
| Online-Zugang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC282402/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2847155/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.85.21.8227 |
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